PART IV: H-M
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a Helmet cells: RBCs that have become folded and damaged, often as a result of the handling of the slide preparation.
Classical darkfield analysis teaches that helmet cells can also appear when dioekothecits have developed within the RBCs and have damaged the membrane when exiting the cell.
When present in plain blood in significant numbers, consider metabolic blockages, intestinal dysbiosis and/or mycosis (fungal infection, ed.), electrolyte insufficiency, reduced enzymatic activity, and lack of sufficient essential fatty acids. (1) (Photograph courtesy of Anna Salanti)
a Infested RBC’s or Parasitized RBCs: cells that are infected with rod forms and embryonic bacteria. They can be both internally parasitized and /or have invisible spindle threads in the membrane. Healthy RBCs will not present this type of degeneration and progression, even when they are isopathically provoked, mechanically stressed, or aged. With this level of infestation, you should explore for catabolic or anabolic imbalance, and consider including intracellular detoxification in the treatment plan. In addition to Mucokehl and Alkala, Vitamin A, Vitamin B, flora inoculation, and spleen glandular may be helpful.(1) (Photograph courtesy of Anna Salanti)
a Lymphocytes: Visual microscopy, whether darkfield, brightfield, or phase contrast, cannot reveal the many varieties and functions of lymphocytes. Specific chemical messengers that interact with specialized cell surface receptors mediate most of the complex tasks these WBCs perform.
The major observations we can make in darkfield are to distinguish between B-lymphocytes and T-lymphocytes based on their size, and to determine whether our DIAD reactions strongly cause lymphocytes to degrade or change in some way.
T-lymphocytes are slightly smaller than RBCs - while B-lymphocytes are about the same size, or a little larger. Each type of lymphocyte should be approximately round, show a well-defined border with no gaps to the large, central nucleus, and should be free of fringes or projections. (1)
(Photograph of T-Lymphocytes courtesy of Anna Salanti)
Macrosymprotits: a large conglomerate of symprotits, which are surrounded by reserve substances and are exceptionally large spheres of purely nucleic protein. They are a cell-wall deficient form that can often be clearly visible in the RBC membranes. It is believed that these intermediate forms are independently related to acute and chronic infection. (1)
a Medusa’s Heads: (See also Radial Tubules) a very clear sign of strong EcoBiotic imbalance. While Naessens describes this as a true fungal form, within the context of DIAD, we have a different theory based on thousands of observations.
In a disturbed field, it is common for many RBCs to develop shiny, bulls-eye centers. While classical hematology attributes this codocyte form to a lack of pigmentation, our observations suggest that in the inner section, where the concave front and back of the RBC are closest, fibers are able to form, eventually fusing the front and back of the corpuscle into a sclerotic, semi-crystalline disk. The RBC then becomes a circular tube around a solid core. When the RBC breaks, the central disk, with attached fibers is freed, forming either a short stranded fila form, or, in more severe cases, the highly developed Medusa’s heads. (1) (Photograph courtesy of Anna Salanti)
Metamychota: (plural) a group encompassing all organisms constructed or assembled out of the cellular units Athrit, Pliathrit, Synathrit or assembled out of Cytit, Pliocytit or Syncytit (protazoans, protophytes, metazoans, metaphytes). (3)
a Microcytes: small, degenerate RBC forms.(Photograph courtesy of Anna Salanti)
Miotrophit: a bacterial cell in a Cyclostage with few (heavily staining) Trophoconies and other nutrients (lipids, nucleic acids, etc.). In stained preparations, it appears less pale than the Atrophit. (3)
Mochlolysis: any cancellation of Mochlosis with metastatic orientation of the resulting Cyclostages. Natural Mochlolysis is that which occurs naturally or due to unknown factors; artificial Mochlolysis is induced by artificial conditional factors. (3)
Monomychota: (plural) the phylum of the Mychoten, encompassing all forms that never proceed beyond Mychit and Diplomychit in their development. Therefore, the formation of Didimychits and Syndimychits never occurs. (3)
Myascit: (See Simple Tubules)
Mych: the proto-nucleus, the carrier of life in the Mychit (the primordial cell). It contains very little to no chromatin and is only slightly more heavily staining (e.g. with fuchsin) than the cytoplasm; with methylene blue mostly non-staining. In the Pliomychit, because of the heavy stainability of the cytoplasm and the yolk elements (reserve materials, Trophoconies) surrounding the Mych, it is usually not possible to make it visible, but its presence and location is indicated by the normally strong clustering of Trophoconies into Trophosoms and Trophosomelles. In the Mychit, especially of the Gonidies, Gonits and atrophic Mychits, as well as Spermit and Oit, it is clearly visible. The diameter is 1/1000 to 1/4000 mm (0.1 - 0.025). (3)
Mychin: the material of the Mych as a physical concept (not chemical). (3)
Mychit: (See Simple Tubules) the primordial cell; a cell with only a single Mych. This includes all individuals of the Monomychoten as well as all monomych reproductive bodies of the Dimychoten (Gonidies, Mychits of the Basits). The Mychit is the morphological base unit of the bacteria and is spherical. (3)
Mychomere: the semivalent proto-nucleus (Mych) of the Gonit (derived from the Gonidie by expulsion and dissolution of the second half of the proto-nucleus) as well as the Spermit and Oit. (3) (See also Spermit.)
Mychota: (plural) kingdom of the Protomychota. (3)
Myscit: (See Simple Tubules)
1. Quoted from and Copyright © 1999 - 2002 Stuart Grace
3. Quoted from introductory glossary to Blood Examination in Darkfield according Prof. Gunther Enderlein, by Dr.Maria M. Bleker
Elements of Comparitive Morphology of Bacteria ©Copyright 1955 for the Estate of Professor Dr. Günther Enderlein, Germany; excerpted from the book, "Bacteria Cyclogeny" by Professor Dr. Günther Enderlein (English version) (Explore Issue: Volume 11, Number 4)